Inhibit的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列問答集和懶人包總整理

The Architecture of Silence: The Abandoned Life of the Italian South

為了解決Inhibit的問題，作者 這樣論述：

During reconstruction of the Italian economy following World War II, the newly established Italian republic and its American allies implemented a program of land reform, the Riforma Fondiaria, which ran from 1950 to 1972. With funding from the Marshall Plan, the Italian state attempted to inhibit

the popularity of the communist party and other left wing movements by appropriating some of their policies. Two extensive re- form laws initiated a redistribution of land that had profound e ects across Italy, albeit predominantly in the south. Nearly 50 years later, what became a spectacular disa

ster for the people and a bonanza for the state has left its physical evidence scattered across the countryside. In 2017, Steven Seidenberg and Carolyn White began an interdisciplinary project to document the contemporary remains of the Riforma. Seidenberg’s richly detailed photographs capture the h

ouses, the outbuildings, the interiors, and the exteriors in a hauntingly beautiful manner, drawing attention to the lives that were strung along through the reform process. Some of the photographs depict the houses themselves, documenting the cast concrete structures posed on the landscape. As Seid

enberg turns his lens toward this rural landscape, he captures the tensions between permanence and temporary, between oc-cupied and abandoned, and where the edge of tolerability exists--places where people moved to live better and where the place was so intolerable that it had to be abandoned again.

Inhibit進入發燒排行的影片

布里斯洛中間體自由基反應機制之理論研究

為了解決Inhibit的問題，作者謝明修 這樣論述：

含氮雜環卡賓（N-heterocyclic carbene）催化之化學反應中，布里斯洛中間體（Breslow intermediate）扮演重要的催化角色。布里斯洛中間體能以親核基（nucleophile）或自由基（radical）之形式參與反應。本論文探討布里斯洛中間體之自由基特性及形成機制（mechanism），其自由基可從氫自由基轉移或直接氧化形成。安息香縮合反應（benzoin condensation）中，布里斯洛中間體將氫原子轉移至苯甲醛（benzaldehyde）以形成自由基，此自由基可結合形成安息香產物，或排除反應之副產物，使其重新進入催化反應。唯此路徑之反應能障高於傳統非自

由基路徑。此研究亦探討四種布里斯洛中間體之不同電子組態的位能面。其中烯醇鹽（enolate）形式能產生偶極束縛態（dipole-bound state），此為產生自由基之新路徑；拉電子基（electron-withdrawing group）以及立體障礙基（bulky groups）可穩定基態。另外，我們亦研究布里斯洛中間體之碎片化（fragmentation）與重組（rearrangement）。布里斯洛中間體之催化反應可能因其碳氮鍵斷裂而中止，形成碎片。我們證實其反應中可以形成自由基，亦可形成離子。反應趨向之路徑與布里斯洛中間體之羥基的質子化型態有關。碎片化反應亦可視為轉酮醇酶（tran

sketolase）中之噻胺（thiamin）催化反應中之副反應；此研究證實轉酮醇酶透過限制布里斯洛中間體之結構與質子化型態，使其碳氮鍵斷裂需更高之反應能量，進而抑制此副反應。

Process!: How Discipline and Consistency Will Set You and Your Business Free

為了解決Inhibit的問題，作者Paton, Mike,González, Lisa 這樣論述：

This deep-dive into the revolutionary EOS method to strengthen a company’s process component will help leaders at all companies--from early stage startups to established corporations--run better businesses and live better lives.Part of the TRACTION Library, Process! proves that a high-level, 20/8

0 approach to getting your core processes ＂followed by all＂ will help you: Get consistently exceptional resultsImprove and innovate as necessary Free yourself to live your ideal lifeIf you own, run, or lead in a fast-moving business, you’re likely driven by passion and a desire to be free. Many lead

ers mistakenly believe instilling rigor and discipline for process throughout your organizations will inhibit freedom. They couldn’t be more wrong. It’s when you’re stuck in the day-to-day, putting out fires and cleaning up messes, that passion turns to frustration. Freedom seems somewhere between e

lusive and impossible. The secret to getting unstuck is process. This inspiring, informative field guide will prove it’s possible to establish rigor and discipline for process while also increasing creativity, flexibility, and innovation. Process! will help you identify a handful of core processes t

hat make your business uniquely valuable. You’ll learn how to document and simplify the major steps in those processes so they can be done the right and best way, every time. Finally, you’ll execute a simple, step-by-step plan that is helping more than 10,000 entrepreneurs around the world consisten

tly get the results they want.

人蔘皂甙 M1 抑制人類口腔癌之效果及作用機轉

為了解決Inhibit的問題，作者理昱傑 這樣論述：

口腔鱗狀上皮細胞癌(Oral squamous cell carcinoma, OSCC)占臺灣所有惡性腫瘤的 5.8%，發病率逐漸上升，為全世界常見的惡性腫瘤，患者存活率極低，因此需要新的有效治療方法來控制口腔鱗狀上皮細胞癌。本篇研究我們製備人蔘皂甙 M1 (20-O--D-glucopyranosyl-20(S)-protopanaxadiol)，為人蔘皂甙主要的去醣基化代謝物，經由真菌 SP-LSL-002 於原料三七葉生物轉化而得，並用於探討人蔘皂甙 M1 於口腔癌細胞及動物模式之抗癌作用及其作用機制。研究結果顯示，人蔘皂甙 M1 可抑制人類口腔癌鱗狀上皮細胞株 SAS 和 OEC

-M1 之存活率。進一步探討人蔘皂甙 M1 之作用機轉，我們發現人蔘皂甙 M1 可增加口腔鱗狀上皮癌細胞之 Bak、Bad 和 p53 蛋白表現，並造成細胞 DNA 斷裂、細胞週期停滯於 G1 期、PI/Annexin V 雙重染色呈現陽性以及 Caspase-3/9 活化，進而促進細胞凋亡。研究結果也證明，人蔘皂甙 M1 可顯著降低 SAS 和 OEC-M1 細胞株之細胞群落生成和遷移能力，並降低癌細胞轉移相關蛋白 Vimentin 之表現。除此之外，以口服或皮下注射給予人蔘皂甙 M1 可明顯抑制 SAS 腫瘤細胞異種移植小鼠之腫瘤生長。綜合以上結果，人蔘皂甙 M1 具有潛力成為口腔癌之治療

藥物。