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infectious gastroent的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列問答集和懶人包總整理
臺北醫學大學 代謝與肥胖科學研究所碩士班 童鈺棠所指導 黃沁兒的 有氧運動對於西式飲食誘導ApoE基因剔除小鼠動脈粥狀硬化之影響 （2019），提出infectious gastroent關鍵因素是什麼，來自於運動、動脈粥狀硬化、腸道菌群、補體因子C5、趨化作用。
而第二篇論文國立臺灣大學 食品科技研究所 潘敏雄所指導 陳佩妤的 探討膳食化合物降低TMAO生成及減緩血管發炎之功效 （2018），提出因為有 動脈粥狀硬化、氧化三甲胺 (TMAO)、肉鹼、腸道菌、膳食化合物的重點而找出了 infectious gastroent的解答。
除了infectious gastroent，大家也想知道這些：
有氧運動對於西式飲食誘導ApoE基因剔除小鼠動脈粥狀硬化之影響
為了解決infectious gastroent 的問題，作者黃沁兒 這樣論述：
Contents摘要 IAbstract IIAcknowledgement IIIContents IVList of tables VIList of figures VIIAbbreviation VIIIChapter 1: Introduction 1Chapter 2: Literature review 32.1 Atherosclerosis 32.1.1 The structure and function of blood vessels 52.1.2 Pathogenesis of atherosclerosis 72.2 Apolipoprot
ein E 92.3 Exercise 12Chapter 3: Effects of aerobic exercise on nonalcoholic liver disease 153.1 Research purpose 153.2 Materials and methods 173.2.1 Animal 173.2.2 Exercise protocol 183.2.3 Blood biochemical measurement 183.2.4 Histopathology analysis 183.2.5 Adipocyte area and adipocyte s
ize distribution 183.2.6 Hepatic steatosis measurement 183.2.7 Western blot analysis 183.2.8 Statistical analysis 193.3 Results 203.3.1 Effects of aerobic exercise on morphology, weight, mean adipocyte size, and distribution of the white adipose tissue 203.3.2 Effects of aerobic exercise on pl
asma hepatic injury indicators and hepatic steatosis 233.3.3 Effects of aerobic exercise on hepatic lipogenesis- and lipolysis-related protein expressions 253.4 Discussion 27Chapter 4 304.1 Research purpose 304.2 Materials and methods 324.2.1 Animal model 324.2.2 Aerobic exercise protocol 34
4.2.3 Plasma biochemical and inflammation analysis 344.2.4 Morphological analysis of atherosclerotic plaque 344.2.5 Fecal microbiota analysis 344.2.6 Fecal short chain fatty acid analysis 364.2.7 Western blot 374.2.8 Proteomic analysis 374.2.9 Immunohistochemical examination 384.2.10 Statisti
cal analysis 394.3 Results 404.3.1 Aerobic exercise alleviates western diet-induced obesity 404.3.2 Effects of aerobic exercise on plasma biochemical parameters 424.3.3 Aerobic exercise ameliorates development of atherosclerotic plaque 444.3.4 Aerobic exercise remodels western-diet-driven dysba
cteriosis of the gut microbiota 464.3.5 Gut microbiota composition with different interventions 504.3.6 Aerobic exercise promotes fecal short chain fatty acid expressions 524.3.7 Aerobic exercise alters microbial-derived short chain fatty acids to suppress chemokine and cytokine expressions
544.3.8 Effects of aerobic exercise on plasma proteomic analysis 584.3.9 Effects of aerobic exercise on complement factor C5 in the aortic root 624.4 Discussion 64Chapter 5: Conclusion 73References 75List of tablesTable 1 Ingredient table of Western diet D12079B 33List of figuresFigure 1 Path
ology of atherosclerosis 4Figure 2 Structure of artery 6Figure 3 Pathogenesis of atherosclerosis 7Figure 4 Apolipoprotein E in lipoprotein metabolism and atherosclerosis 10Figure 5 Physical activity promotes cardiorespiratory fitness 14Figure 6 Effects of aerobic exercise on morphology, weight,
mean adipocyte size, and adipocyte distribution 21Figure 7 Effects of aerobic exercise on plasma hepatic injury indicators and hepatic steatosis 24Figure 8 Effects of aerobic exercise on hepatic lipogenesis- and lipolysis-related protein expressions 26Figure 9 Effects of aerobic exercise on food
intake, caloric intake, body weight and feed efficiency ratio 41Figure 10 Effects of aerobic exercise on biochemical variables 43Figure 11 Effects of aerobic exercise on pathogenesis of atherosclerosis 45Figure 12 Effects of aerobic exercise on taxonomic differences in gut microbiota population
48Figure 13 Effects of aerobic exercise on gut microbiota community 49Figure 14 Effects of aerobic exercise on gut microbiota composition at genus level 51Figure 15 Effects of aerobic exercise on gut microbiota-derived SCFA 53Figure 16 Effects of aerobic exercise immune cell infiltration in cho
lesterol plaque formed in aortic root 56Figure 17 Effects of aerobic exercise on aorta chemokines and proinflammatory protein expressions 57Figure 18 Effects of aerobic exercise on plasma proteomic analysis 60Figure 19 Effects of aerobic exercise on cardiovascular and hematological diseases regul
ated proteins 61Figure 20 Effects of aerobic exercise on complement factor C5 in the aortic root 63Figure 21 Possible effects of aerobic exercise on pathogenesis of atherosclerosis 7
探討膳食化合物降低TMAO生成及減緩血管發炎之功效
為了解決infectious gastroent 的問題，作者陳佩妤 這樣論述：
據統計，心血管疾病為全球人口最常見的的發病原因，並且是全世界十大死因的第一名。動脈粥狀硬化是造成心血管疾病的主要原因，已被視為工業化國家的公共衛生問題。 近年來，氧化三甲胺 (trimethylamine-N-oxide, TMAO)被認為會導致動脈粥狀硬化，並與心血管疾病有很強的相關性。TMAO是由膳食中的肉鹼(carnitine)經由腸道菌代謝成三甲胺 (trimethylamine, TMA)，隨後再經宿主的肝臟酵素flavin monooxygenases (FMOs)氧化成TMAO。本研究的目的是想探討多酚類化合物 (pterostilbene, oolong tea extrac
t and PMFs)對於TMAO生成及減緩血管發炎之效果。結果顯示，相較於控制組，單獨給予1.3 %肉鹼飲用水的組別顯著性的增加了血漿中TMAO的含量；而給予多酚類化合物的小鼠，則可以降低肉鹼所引起的血漿中TMAO上升的現象。此外，相較於單獨給予肉鹼的組別，給予多酚類化合物的小鼠，其肝臟酵素FMO3的mRNA表現量都有顯著性的下降。採集動脈進行血管發炎因子 (TNF-α, VCAM-1 and E-selectin) mRNA表現量測定，結果顯示給予本篇使用的多酚類化合物之小鼠可以減緩血管的發炎。相較於單獨給予肉鹼的組別，PMFs顯著地降低了VCAM-1 mRNA表現量，而給予pterost
ilbene以及烏龍茶萃取物的組別則顯著性降低了TNF-α, VCAM-1和E-selectin mRNA表現量。經腸道菌分析結果顯示，三種多酚類化合物的給予都增加了類桿菌屬的相對豐富。而給予烏龍茶萃取物的組別，則可增加乳桿菌屬的相對豐富。艾克曼菌 (Akkermansia)則是在給予polymethoxyflavones的組別有觀察到增加的現象。綜合以上結果，此三種多酚類化合物可以經由調節腸道菌相以及降低肝臟酵素FMO3而達到減緩血管發炎的作用。